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Non-Smad pathways in TGF-beta signaling

Mohammed Vashist, N

10.1089/ten.2007.0101 50

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Subsequently, the authors introduced a new method for treating gastroschisisperforming a one-stage reduction of intestinal loops after emptying the intestines through the rectum and the opened lumen of the appendix

When we quantified the levels of inflammatory mediators that have previously been shown to induce BBB dysfunction [42, 47, 58], Tnf (tumor necrosis factor), Il1b (interleukin 1 beta), and Ccl2 (C-C Motif Chemokine Ligand 2) were strongly upregulated (Fig