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These findings suggest that targeting 7nAChR with specific agonists may hold potential for treating cognitive impairments in ASD [35, 73, 74]

Relocalized redox-active lysosomal iron is an important mediator of oxidative-stress-induced DNA damage

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Our previous work demonstrated the presence of cells containing SGs adjacent to the hypoxic GBM core in vivo, in human GBM samples suggesting a role in GBM pathobiology [18]

Ferroptosis is a unique iron-dependent form of non-apoptotic cell death, characterized by lipid peroxidation with high levels of glutathione consumption

Standards or samples are added to the appropriate microtiter plate wells then with a biotin-conjugated antibody specific to Human IFNb